Oxycontin Addictions

oxycontin addictions

Alcohol is one teratogen that could greatly affect the woman’s pregnancy. People may not know of its danger and still allow a woman to drink alcoholic drinks during her pregnancy, but the result of this would be carried by the baby for the rest of his or her life. In the United States, alcohol is the substance very famous to cause mental and physical birth defects. Though, this is only a probability, the rate is high.

For one, Fetal Alcohol Syndrome is a disorder that can occur to the embryo when a pregnant woman ingests alcohol during pregnancy. Alcohol crosses the placental barrier and can inhibit fetal weight, create distinctive facial stigmata, damage neurons and brain structures, and cause other physical, mental, or behavioral problems. The main effect of FAS is stable central nervous system damage, especially to the brain. Developing brain cells and structures are malformed by before birth alcohol exposure, often creating a collection of primary cognitive and functional disabilities including poor memory, attention deficits, impulsive behavior, and poor cause-effect reasoning, as well as secondary disabilities for example, mental health problems, and drug addiction.

The signs and symptoms of a having fetal alcohol syndrome are low birth weight, small head circumference, developmental delay, organ dysfunction, facial abnormalities, including smaller eye openings, flattened cheekbones, and indistinct philtrum (an underdeveloped groove between the nose and the upper lip), epilepsy, poor coordination, poor socialization skills, such as difficulty building and maintaining friendships and relating to groups, lack of curiosity, learning difficulties, including poor memory, inability to understand concepts such as time and money, poor language comprehension, poor problem-solving skills, behavioral problems including hyperactivity, inability to concentrate, social withdrawal, stubbornness, impulsiveness, and anxiety.

As said earlier, damage of the central nervous system is the main feature of fetal alcohol syndrome. Central nervous system damage can be assessed in three areas such as structural, neurological, and functional deficiencies. Structural deficiencies includes microcephaly (small head size) of two or more standard deviations below the average, or other abnormalities in brain structure. During the first trimester of pregnancy, alcohol gets in the way with the migration and organization of brain cells, which can create structural deficits within the brain. During the third trimester, harm can be caused to the hippocampus, which plays a role in memory, learning, emotion, and encoding visual and auditory information, all of which can create neurological and functional CNS impairments as well.

When structural impairments are not observable, neurological impairments are assessed. Neurological problems are showed as either diagnosable disorders, such as epilepsy or other seizure disorders, or soft signs. Soft signs are broader, nonspecific symptoms, such as impaired fine motor skills, neurosensory hearing loss, poor gait, clumsiness, poor eye-hand coordination.

Functional impairments are assessed when structural or neurological impairments does not exist. Functional impairments are problems due to prenatal alcohol exposure (rather than hereditary causes or postnatal insults) in observable and measurable domains related to daily functioning, often referred to as developmental disabilities.

The production of birth deficiencies is not a measure on the amount of alcohol being ingested. Individual women process alcohol differently. Other factors differ the results, too, such as the age of the mother, the timing and regularity of the alcohol ingestion, and whether the mother has eaten any food while drinking.

Alcohol quickly passes the placental barrier and the fetus is less equipped to get rid of alcohol than its mother, so the fetus tends to accept a high concentration of alcohol, which lingers longer than it would in the mother’s system. That explains why the child is the one greatly affected when in fact it is the mother who consumes the alcohol. This such a sad truth.

Oxycontin Addictions

oxycontin addiction

Oxycontin Addictions

oxicontin addiction

Oxycontin Addictions

oxycontin addiction

Depending upon the preference of the user and the drugs’ purity. Heroin can be used in different ways. Heroin can be injected into a vein, injected into a muscle, smoked in a standard pipe, mixed in a regular cigarette, inhaled as smoke through a straw, known as “chasing the dragon,” snorted as powder via the nose.

The short-term effects of heroin abuse appear soon after a single dose and disappear in a few hours. After an injection of heroin, the user reports feeling a surge of euphoria accompanied by a warm flushing of the skin, a dry mouth, and heavy extremities. The initial euphoria is followed by an alternation of a wakeful and sleepy state. Mental functioning becomes clouded due to the depression of the central nervous system. Other effects included slowed and slurred speech, slow gait, constricted pupils, droopy eyelids, impaired night vision, vomiting, constipation.

A certain period of time is needed for the appearance of the long-term effects of heroin. Chronic users may develop collapsed veins, infection of the heart lining and valves, abscesses, cellulites, and liver disease. Pulmonary complications, including different types of pneumonia, may result from the poor health condition of the abuser, as well as from heroin’s depressing effects on respiration. In addition to the effects of the drug itself, street heroin may have additives that do not really melt and result in clogging the blood vessels that lead to the lungs, liver, kidneys, or brain. This can cause infection or even death of small patches of cells in vital organs. Tolerance develops with regular use of heroin. This means the abuser must use more heroin to achieve the same intensity or effect.

As higher doses are used over time, physical dependence and addiction develop. With physical dependence, the body has adapted to the presence of the drug and withdrawal symptoms may occur if use is reduced. Withdrawal, which in regular abusers may happen as early as a few hours after the last administration, produces drug craving, restlessness, muscle and bone pain, insomnia, diarrhea and vomiting, cold flashes with goose bumps (”cold turkey”), kicking movements (”kicking the habit”), and other symptoms. Major withdrawal symptoms hit the highest point between 48 and 72 hours after the last does and subside after about a week. Sudden withdrawal by heavily dependent users who are in poor health can be deadly.

Furthermore, the withdrawal syndrome from heroin may begin within 6 to 24 hours of discontinuation of the drug; however, this time frame can vary with the degree of tolerance as well as the amount of the last consumed dose. Symptoms may include: sweating, malaise, anxiety, depression, priapism, extra sensitivity of the genitals in females, general feeling of heaviness, cramp-like pains in the limbs, excessive yawning, tears, rhinorrhea, sleep difficulties (insomnia), cold sweats, chills, severe muscle and bone aches; nausea and vomiting, diarrhea, cramps, and fever.

Heroin addiction, just like any other drug addiction starts with occasional use of the drug, which later on would become a habit. This is so because it causes some chemicals in the brain to become dependent to the drug. Moreover, the body itself desires for the drug because of its pleasurable effects. It is relaxing, thus the person feels positively about the drug. And even though it gets to the point that the drug is slowly destroying the person, he or she could not get away with it anymore, because he or she has become dependent on heroin. Thus, when the drug intake is suddenly stopped, the person experiences withdrawal symptoms. With this, professional help is needed for recovery.

Oxycontin Addictions

oxycotin addiction

Oxycontin Addictions

oxycontin addictions

Oxycontin Addictions

oxicontin addiction

Oxycontin Addictions

oxycontin addiction